NURSING INTERVENTION FOR THE PREVENTION OF TORSADE DE POINTES IN PATIENTS WITH LONG QT SYNDROME
Section: Artículos Científicos
How to quote
Enferm Cardiol. 2018; 25 (74): 40-50.
Authors
Miguel Ángel Andrés Gasco
Position
Enfermero UCI Polivalente Hospital Universitario Príncipe de Asturias.Experto en Urgencias y emergencias extrahospitalarias.Graduado Universitario en Enfermería y Criminología.Proveedor de SVCA/ACLS por la AHA y SEMES.
Contact address
Miguel Ángel Andrés Gasco. C/ José Demétrio Calleja, 3 28804. Alcalá de Henares Madrid
Abstract
During healthcare practice, nursing professionals use a wide variety of drugs with different properties and adverse effects. In this article, we have wanted to focus on those drugs that generate a QT prolongation, and especially in those patients with a Long QT Syndrome, the administration of which can give rise in many cases to the appearance of arrhythmias such as Torsade de Pointes.
Keywords:
Torsade de Pointes; channelopathies; medication errors; patient safety
Versión en Español
Título:
Artículo completo no disponible en este idioma / Full article is not available in this language
Bibliography
- Jervell A LNF. Congenital deaf-mutism, functional heart disease with prolongation of the Q-T interval and sudden death. Am Heart J. 1957.
- Amin A AATH. Cardiac sodium channelopathies. Pflugers. 2010.
- Bennett PB YKMNea. Molecular mechanism for an inherited cardiac arrhythmia. Nature. 1995.
- Ackerman MJ MP. Defining a new paradigm for human arrhythmia syndromes:phenotypic manifestations of gene mutations in ion channel- and transporter-associated proteins. Circ Res. 2010.
- Schwartz PJ PSSCMAVGNCea. Genotype-phenotype correlation in the long-QT syndrome: gene-specific triggers for life-threatening arrhythmias. Circulation. 2001.
- Ackerman MJ TDPC. Swimming, a gene-specific arrhythmogenic trigger for inherited long QT syndrome. Clin Proc. 1997 Mayo.
- Anderson CL, Delisle BP, Anson BD, et al. Most LQT2 mutations reduce Kv11.1 (hERG) current by a class 2 (trafficking-deficient) mechanism. Circulation. 2006;113:365–373.
- Beaufort-Krol GC VdBMWAVTJVJBCea. Developmental aspects of long QT syndrome type 3 and Brugada syndrome on the basis of a single SCN5A mutation in childhood... J Am Coll Cardiol. 2005.
- Mohler PJ SJGADKGSdWea. Ankyrin-B mutation causes type 4 long-QT cardiac arrhythmia and sudden cardiac death. Nature. 2003.
- Mohler PJ SINCBGSLTKea. A cardiac arrhythmia syndrome caused by loss of ankyrin-B function. Proc Natl Acad Sci USA. 2004.
- Splawski I TFMLMSMKM. Mutations in the hminK gene cause long QT syndrome and suppress IKs function. Nat Genet. 1997.
- Abbott GW SFSIBMLMTKea. MiRP1 forms IKr potassium channels with HERG and is associated with cardiac arrhythmia. Cell. 1997.
- Splawski I TKSLDNKPBRea. Ca(V)1.2 calcium channel dysfunction causes a multisystem disorder including arrhythmia and autism. In.; 2004. p. 119.
- Curran ME SITKVGGEKM. A molecular basis for cardiac arrhythmia: HERG mutations cause long QT syndrome. cell. 1995.
- Medeiros A KTTDIPIAYBea. Sodium channel B4 subunit mutation causes congenital long QT syndrome. Heart Rhythm. 2006.
- Neyroud N TFDILMDCBJea. A novel mutation in the potassium channel gene KVLQT1 causes the Jervell and Lange-Nielsen cardioauditory syndrome. Nat Genet. 1997.
- Schwartz PJ SCCLBJAJTKea. The Jervell and Lange-Nielsen Syndrome. natural history, molecular basis, and clinical outcome. Circulation. 2006.
- Moss AJ , Zareba W , Benhorin J , et al. ECG T-wave patterns in genetically distinct forms of the hereditary long QT syndrome. Circulation. 1995;92:2929-2934.
- Schwartz PJ MAVGCR. Diagnostic criteria for the long QT syndrome. An update. Circulation. 1993; 88(782-4).
- Zhang L BDTFMPLTRSPea. Electrocardiographic features in Andersen-Tawil syndrome patients with KCNJ2 mutations: characteristic T-U-wave patterns predict the KCNJ2 genotype. Circulation. 2005.